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ROS-activated CXCR2+ neutrophils recruited by CXCL1 delay denervated skeletal muscle atrophy and undergo P53-mediated apoptosis

Medicine and Health

ROS-activated CXCR2+ neutrophils recruited by CXCL1 delay denervated skeletal muscle atrophy and undergo P53-mediated apoptosis

Y. Xiang, J. Dai, et al.

Discover how reactive oxygen species (ROS) activate neutrophils to combat muscle atrophy following denervation in this groundbreaking research by Yaoxian Xiang and colleagues. Their study reveals a potential therapeutic target in neutrophils for treating skeletal muscle atrophy, presenting exciting new avenues for intervention.

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