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Celastrol suppresses neovascularization in rat aortic vascular endothelial cells stimulated by inflammatory tenocytes via modulating the NLRP3 pathway

Medicine and Health

Celastrol suppresses neovascularization in rat aortic vascular endothelial cells stimulated by inflammatory tenocytes via modulating the NLRP3 pathway

Y. Yang, H. Wang, et al.

Discover how inflamatory processes in tenocyte injury impact angiogenesis during tendon-bone healing. This research, conducted by Yong Yang and colleagues, reveals that Celastrol can suppress excessive angiogenesis, leading to improved tendon healing outcomes.

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